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2019年1月 2日 (水)

colonizationとか、colonizeとか

医学翻訳フリーランサーのみなさん、こんにちは。

Medical Translator NARITAです。

 

さて、

colonizationとか、colonizeとか。

なんとなくわかるが、いざ訳そうとすると迷います。

 

ライフサイエンス辞書では、

colonization

コロニー形成,    細菌叢,    集落形成,    (細菌の)   定着

コロニー形成」を訳語にするとしっくりこない。

 

コロニー形成」は実験室での細菌などの培養で使う用語のようです。

https://ja.wikipedia.org/wiki/%E3%82%B3%E3%83%AD%E3%83%8B%E3%83%BC%E5%BD%A2%E6%88%90%E5%8D%98%E4%BD%8D

臨床系の文書で「コロニー形成」がしっくりこないのはそのためかもしれません。

 

ロゼッタさんの究極の辞書で調べましたが、どうなんでしょう。

https://www.sangyo-honyaku.jp/dictionaries/index/search_info:%E3%82%B3%E3%83%AD%E3%83%8B%E3%83%BC%E5%BD%A2%E6%88%90%E7%8E%87_%E9%9D%9E%E8%87%A8%E5%BA%8A

 

とても参考になる解説が見つかりました。

http://www.wound-treatment.jp/wound014.htm

日本語の臨床系の文書ではInfectionとColonizationを厳密に区別していないような印象があります。

以上の情報に基づき、イートモではcolonizationとcolonizeの和訳を整理しなおしました。

 

イートモ対訳データベースからcolonizationまたはcolonizeを含む対訳を抽出してご参考まで提示しました。ちゃんと表示するしかたがわからないので、和文の右端が切れてしまってすみません。

なお、イートモ対訳は随時修正しています。

必要ならば速攻で修正します。

ですから、イートモユーザー様は他のツールにコピペせず、なるべくイートモシステム内で利用してください。

http://i-honyaku.cocolog-nifty.com/blog/2018/10/pdicebwin4-a2b8.html

 

A person colonized with a cag+, s1a   vacA, iceA1 strain is more likely to develop duodenal ulceration than a   person harboring a cag, s2 vacA, iceA2 strain.
Bifidobacterium bifidum is the predominant   bacterial species in the intestine of breast-fed infants, where it presumably   prevents colonization by potential pathogens.
Central venous catheters coated or   impregnated with antimicrobial agents effectively prevent microbial   colonization and catheter-related bloodstream infections.
Colonization induces chronic gastric   inflammation which can progress to a variety of diseases.
Helicobacter pylori colonizes the   gastric mucosa, where it appears to persist throughout the host's life unless   the patient is treated.
Helicobacter pylori is a gram-negative organism that is able to colonize the human   stomach.
If a suitable vaccine were   available (research projects are ongoing) to prevent H pylori colonization,   would we increase or decrease the disease burden?
It is likely that H. pylori has colonized our stomachs since well before we   became humans.
It is possible to acquire infection   from a partner with genital or oral colonization.
Large numbers of people are now passing their lives without H. pylori   colonization.
Little is known, however, about   their significance in oral colonization and cariogenicity.
Lymphoid proliferation is a common feature of H. pylori   colonization. 
Monitoring trends in colonization   rates should provide clues.
Much of the recent work in tackling methicillin-resistant   Staphylococcus aureus (MRSA) has focused on hygiene in hospitals, but it is   unclear how much hospital staff know about the treatment and management of   patients who are colonized or infected with MRSA.
Normal human skin is colonized with   bacteria; different areas of the body have varied total aerobic bacterial   counts.
Once S. aureus successfully adheres to and colonizes host tissues,   the expression of specific genes is altered, resulting in a phenotype that is   more resistant to antibiotics.
One explanation is that the stomach   appears to be colonized by different organisms than those in patients with   hypochlorhydria.
People carrying H. pylori may be slightly shorter (and possibly   leaner) than their uncolonized counterparts.
Peptic ulcer disease and gastric   cancer have been declining in the 20th century in precisely those parts of   the world in which the prevalence of H. pylori colonization has declined.
The hands of health care workers may become   persistently colonized with pathogenic flora (e.g., S. aureus), gram-negative   bacilli, or yeast.
The   microorganism colonizes the nose and paranasal sinuses, spreading into   adjacent tissues by invading blood vessels.
The prevalence of H. pylori colonization is about 30% in the United States   and other developed countries as opposed to > 80% in many developing   countries.
The   published literature on this is often difficult to interpret because of   heterogeneity in the density of bacterial colonization and in host immune   response.
The reasons for the decline in the prevalence of H. pylori colonization   are unknown.
The   strain defective in ABC could not colonize teeth of starch-only-eating rats.
These valves then become colonized by bacteria of low virulence.
Transient flora, which colonize the   superficial layers of the skin, are more amenable to removal by routine hand   washing.
With continuing socioeconomic development, H pylori colonization is   becoming less common.

 

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